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| Available: 0 |
| List Price:$185.00 |
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| Edition: | 1st |
| Publisher: | Oxford University Press, Incorporated |
| ISBN: | 0-19-509483-2 (0195094832) |
| ISBN-13: | 978-0-19-509483-1 (9780195094831) |
| Binding: | Hardcover |
| Copyright: | 2007 |
| Publish Date: | 04/07 |
| Weight: | 2.50 Lbs. |
| Pages: | 720 |
| Carton Quantity: | 11 |
| Subject Class: | CAR (Cardiology) |
| Remarks: | Print on demand. If not in stock, allow additional time for processing |
| Doody Star Rating: |
3 |
| Return Policy: | Returns accepted up to 90 days provided no other recalls or return restrictions apply. |
| Table Of Contents: |
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| Contributing Authors: |
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| Discipline: | Cardiovas Sys | | Subject Definition: | Hypertension-Etiology | | NLM Class: | WG 340 | | LC Class: | RC685 | | Abstract: | Essential hypertension (EH) is the most common type of high blood pressure and is responsible for much death and serious illness, which has been the reason for the huge research effort to determine its causes. However, countless scientific articles still keep proclaiming that "the causes of essential hypertension remain unknown." In view of the number of publications that descend on the hypertension specialist like a waterfall, such proclamations seem to overlook the considerable amount of knowledge that we already have about the etiology of EH. The problem may be a lack of synthesis rather than a lack of information. This book brings together some of this knowledge into a coherent and clinically relevant account about the pathogenesis of EH. It presents a new theory about the causes of essential hypertension (EH), where the brain initiates the rise in blood pressure (BP). Chronic stress is crucial for the development of EH, with the hypothalamic hyperresponsiveness due to the strengthening of a synapse linking cortex with hypothalamus. Additional chapters compare the development of high BP in renal hypertension and in the Japanese Spontaneously Hypertensive Rat with that of EH. |
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